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In animal models, GLP-1 enhances myocardial glucose usage, shifting metabolism from fatty acid to carbohydrate oxidation

Additional observations from Phase 2 data include: Mild increases in heart rate, attributed to the glucagon receptor component

If the therapy has been interrupted for a long time, the body is no longer adapted to the physiological changes caused by the medication

1 Metabolic diseases like obesity and overweight have traditionally been treated with combinations of behavioral therapy and pharmacotherapy, the latter of which was often associated with poor efficacy and weight regain after medication cessation

Tirzepatide for heart failure with preserved ejection fraction and obesity

This decrease could encourage osteoblast development and differentiation, which would benefit the skeleton (69)