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These findings underscore the potential for GLP-1 RAs to offer tailored protection against complex diabetic complications, marking an advancement in personalized medicine for managing diabetes-acute kidney disease.

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Twenty-nine studies were reviewed with follow-up ranging from 1 to 22 years

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The authors found that the number of GLP-1 expressing cells is consistently reduced at the site of active disease in both mouse models of intestinal inflammation and in CD patients, as confirmed by the single-cell RNA sequencing data