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Disruptions in homeostasis from quick weight reduction can elevate cortisol and norepinephrine levels, heightening the risk for, or exacerbation of STB

Since GLP-1RAs simultaneously target IR, lipid profiles, and hepatic fat, they provide an actionable pharmacological tool for risk-forward preventive strategies (78, 83)

Their findings were striking: AKD patients treated with GLP-1 RAs experienced lower rates of major adverse kidney events (MAKEs), which include complications like the need for dialysis, worsening kidney function, and even death due to kidney failure

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Every patient is evaluated by a licensed physician or nurse practitionernever an automated system

Yet, GLP-1 is mainly localized to the distal small bowel and colon and is released following nutrient intake [12, 13] and it has been shown that M1 and M2 muscarinic receptors may also be involved in the regulation of GLP-1 release [14]