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The future perspectives would include different doses of DPPIV-inhibitors would need to be administered for different levels of endogenous GLP-1

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However, in highly invasive breast cancer cell lines MDA-MB-231 and MDA-MB-468, high concentrations (100 nM) of liraglutide promote breast cancer progression by activating GLP-1R via the NOX4/ROS/VEGF signaling pathway [18]