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This reduced involvement may stem from factors such as embarrassment, anticipated pain during treatments, or previous negative healthcare experiences [6]

In the center, leucine is metabolized to 3-methylcrotonyl-CoA (via BCAA aminotransferase and branched-chain -ketoacid dehydrogenase) and then via the biotin-requiring methylcrotonyl-CoA carboxylase to 3-methylglutaconyl-CoA, 3-hydroxy-3-methylglutaryl-CoA, and then to acetoacetate and acetyl-CoA

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Lipid dyshomeostasis interacts with several AD pathogenic pathways, including amyloidogenesis, mitochondrial dysfunction, oxidative stress, neuroinflammation, and myelin degeneration (Yin, 2023

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