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In a related study, liraglutide was shown to increase FVC, and carbon monoxide diffusion capacity, and significantly reduce serum surface active protein D (SP-D) levels (used as a biomarker to assess alveolar-capillary barrier integrity) in patients with type 2 diabetes [48, 49]

Specialized localization of P-glycoprotein recognized by MRK16 monoclonal antibody in endothelial cells of the brain and the spinal cord

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Collectively, these data suggest that voluntary cocaine taking activates GLP-1-producing neurons in the NTS through a corticosterone-mediated mechanism of action and that increased GLP-1 receptor signaling in midbrain and forebrain areas represents a negative-feedback response that reduces the reinforcing efficacy of cocaine

Interestingly, GLP-1 is also produced directly in the CNS by preproglucagon neurons, predominantly in the brainstem (DAlessio, 2016