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These effects involve GLP-1Rs in the NAc shell and central CeA and are likely mediated in part by modulation of stress and anxiety circuits

This phenomenon refers to increased stimulation of insulin secretion by the pancreas after oral glucose ingestion compared to intravenous glucose infusion

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It reversed glucose dysregulation without detrimental effects on metabolically healthy animals and reduced body weight, lowered fasting blood glucose, decreased glycosylated hemoglobin (HbA1C), improved glucose tolerance, and protected pancreatic islet architecture in diabetic fatty Zucker rats 14,19,20

I also appreciate that they have the resistance weight printed on them