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These initial neurological changes occur as semaglutide binds to GLP-1 receptors in the hypothalamus, signaling satiety before physical weight reduction occurs

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When SCFAs, a metabolite of GM, was supplemented to antibiotic-treated mice, IGF-1 and bone mass were restored to the levels of normal mice, suggesting that the restoration of the GM in germ-free mice is mediated by the production of SCFAs, which induces circulating levels of IGF-1 and thus has an effect on bone metabolism (Yan et al., 2016)

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