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In vivo , the GIP(5-31) analogue inhibited GIP-stimulated insulin secretion in human islets

Iwai T, Ito S, Tanimitsu K, Udagawa S, Oka J-I

Over time, this energy shortfall sets off a cascade of downstream effects: synaptic dysfunction, impaired protein clearance, neuroinflammation, and ultimately the accumulation of amyloid- and tau pathology Importantly, imaging and metabolic studies consistently show that reductions in cerebral glucose metabolism and perfusion precede cognitive symptoms by years, often decades [8]

Brain Sci

The number needed to treat to prevent one major adverse cardiovascular event after one and three years of use was 221 and 86, respectively

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