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1.3.9 H3K9 demethylases H3K9me2 is a substrate for KDM4A and PHF8

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(vii) inhibition of hepatic apoptosis

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111 Reduced production of pro-inflammatory cytokines, blocked NF-B signaling, and increased regulatory T-cell activity are some of the mechanisms through which GLP-1RA protects against intestinal inflammation in early-stage IBD models 112 (Figure 1)