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All of this happens on a weekly rhythm

(NCBI) Think of GLP-1 signalling as an appetite brake + pacing system: Brain: appetite and reward modulation Gut: slower gastric emptying Pancreas: insulin secretion (glucose-dependent) and reduced glucagon secretion (context-dependent) The critical factor is that the lack of appetite typically leads to a caloric deficit

Not everyone with NAFLD will experience the same liver benefit, so physicians must monitor individual outcomes and modify strategies accordingly

When blood glucose remains persistently high, as it does in poorly managed diabetes, it causes microvascular damage throughout the body

Occasionally, non-diabetics will have a negative gastric emptying study and no evidence of delayed emptying, but when a liquid gastric emptying study is done it will be abnormal

Telogen effluvium typically resolves within 36 months once the triggering factor stabilises