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To evaluate whether the difference in response between females and males was attributed to sex differences in semaglutide dose or in type 2 diabetes status, we further examined BMI trajectories only among patients without type 2 diabetes who reached a semaglutide dose 1.7 mg

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These include model phenotyping studies and the characterization of several MASH-targeted drug classes, such as GLP1R agonists (e.g., semaglutide), THR- receptor agonists (e.g., resmetirom), PPAR agonists (e.g., lanifibranor), FGF21 analogues (e.g., efruxifermin), as well as various long-acting dual GLP1R-GIPR agonists (e.g., tirzepatide) and GLP1R-GCGR agonists (e.g., survodutide)