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These studies have revealed that the specific composition of the GM contributes to the specific response of its host (i.e

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This unique presentation of both NAWE and TIND in a single patient following GLP-1 agonist use warrants careful consideration of the potential central and peripheral neurological sequelae of these increasingly prevalent medications and emphasizes the need for heightened clinical awareness and appropriate monitoring strategies

The proposed mechanisms of diabetes-related NDs include cerebral insulin resistance (IR), vascular endothelial dysfunction, inflammation, bloodbrain barrier injury, white matter disease of vascular origin, demyelination and axonal loss, and peroxidative membrane injury.11 Among these mechanisms, brain IR may play a primary role, and it is worth noting that neurologic complications may already occur with prediabetes IR.12 Oxidative stress,13 mitochondrial dysfunction,14 and endoplasmic reticulum (ER) stress15 are all involved in NDs induced by brain IR

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