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Semaglutide works by activating GLP-1 receptors in the brain's appetite centers, creating feelings of fullness and reducing hunger-driven eating

Does my genetics suggest a different GLP-1based treatment might be better suited

retina specialists treating very severe NPDR without DME, 60% closely monitor the condition and encourage systemic glycemic control, 25% consider anti-VEGF therapy in some patients with poor glycemic control, around 8% consider it in all or most patients, and 3% consider it in some patients with good glucose control and compliance

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This does not mean the exposure was risk-free, but it means outcomes are often good despite imperfect circumstances

In any case, Mayo Clinic confirms that all GLP-1 agonists tend to produce weight loss , and that tirzepatide (and semaglutide) are generally the most potent in trials