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In addition, the gut-derived metabolite imidazole propionate has been shown to drive -synuclein pathology in preclinical and patient-derived PD models, providing a direct link between dysbiosis and -synuclein aggregation (103)

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NAMPT-overexpressing cells, either in the mass culture or tumorspheres, were more sensitive to FK866 than were parental cells or cells with reduced levels of NAMPT

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