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10.3389/fnmol.2022.908948 68 TuestaL

[DOI] [PMC free article] [PubMed] [Google Scholar] 8.Bang-Berthelsen CH, et al

Exenatide treatment ameliorates these morphological alterations and inhibits the mitochondrial apoptotic pathway by suppressing cytochrome c release and favorably modulating the expression of pro-apoptotic (Bax, caspase-3) and anti-apoptotic (Bcl-2) proteins, thereby protecting RGC-5 cells from hyperglycemic injury [43]

Based on these reports, the impairment of synaptic function and synaptic transmission and the loss of synaptic density proteins are widely observed in the depressive brain ( Figure 2 )

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Transition 3: morphs between the three major classes of location of the Gs AHD for the semaglutide and taspoglutide bound GLP-1R-Gs complexes determined from focused 3D classification in RELION