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Using a combination of chemogenetic, optogenetic, and pharmacological methods in wild-type and Glp-1Rdeficient mice, the authors delineated an NTS MHb IPN pathway responsible for nicotine avoidance

doi: 10.1073/pnas.89.18.8641

Tapering semaglutide to the most effective dose: real-world evidence from a digital weight management programme (TAILGATE)

These data indicate that the disulfide bonds of both E127 ECD CQ211 ECL1 C and Q37 ECD CL379 ECL3 C are formed on the native protein in the absence of the antibody, strengthening the hypothesis that the current structure represents a physiologically relevant inactive conformation of GLP-1R