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doi: 10.3390/cells11071187

Nutrient timing and mineral balance can also influence muscle preservation during caloric restriction, particularly in the context of magnesium-dependent metabolic pathways

The use of GLP-1R agonists, glucagon agonists, GLP-1glucagon dual agonists (DualAG), GIPGLP1glucagon receptor agonists, and GLP1GIP agonists has led to the premise that incorporating glucagon receptor agonists reduces energy intake, increases energy expenditure, or potentially enhances efficacy [18]
