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Given the established clinical use of GLP-1RAs for the treatment of T2DM, preclinical and clinical research was centered largely around DR, which suggested the neuropathologic protective effects of GLP-1 are due to downregulation of NF-B, proinflammatory cytokines such as TNF-alpha and IL-1beta, and VEGF

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Tissue Histology Mouse tissues were fixed in 10% neutral buffered formalin (NBF) for hematoxylin and eosin (H&E) staining or in 1% periodic acid prepared in 10% NBF for periodic acidSchiff (PAS) staining

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The increase in heart rate by teratrutide could be mediated in part by glucagon receptor agonism