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41 The significant reduction in urinary oxalate excretion after weight loss through GLP-based therapies could possibly be attributed to decreased intake of oxalate and oxalate precursors or to reduced hepatic oxalate synthesis

This outcome is thought to be an effect of all medications that increase GLP-1, because of both an increase in SDF-1-alpha and capillary density due to increased hypoxia-inducible factor 1-alpha and vascular endothelial growth factor

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It also helps ease common digestive discomforts often associated with GLP-1 protocols including occasional bloating, gas, and digestive irregularities

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These drugs are essential to prevention of rejection but come at a metabolic cost, disrupting glucose metabolism through several mechanisms, including downregulation of transcription of the adiponectin gene which can lead to greater risk for glucose intolerance