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Although it appears surprising that the intracellular [Ca 2+ ] response to GABA was almost completely abolished by the specific GABA A receptor antagonist bicuculline, despite the evidence suggesting that GABA C receptor activation contributes to the GABA response, similar findings in neurones have recently led to the proposal that GABA A and GABA C receptor subunits can coassemble to form heteromeric channels with mixed properties (Hartmann et al

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1 Department of Biomedicine, Aarhus University and Steno Diabetes Center Aarhus, Aarhus, Denmark, 2 Clinic for General, Visceral, and Pediatric Surgery, University Medical Center Gttingen, Gttingen, Germany, 3 Department of Cardiovascular Sciences, Center for Molecular and Vascular Biology, KU Leuven, Leuven, Belgium, 4 Division of Cardiovascular Medicine, Department of Medicine Solna, Karolinska Institute, Stockholm, Sweden, 5 Department of Biochemistry and Molecular Cell Biology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany, 6 Department of Biomedicine, Aarhus University, Aarhus, Denmark, 7 Division of Endocrinology, Diabetes and Metabolism, Beth Israel Deaconess Medical Center, Boston, MA, USA, 8 Department of Medicine Huddinge (H7), Karolinska Institute, Stockholm, Sweden, 9 Helmholtz Institute for Metabolic, Obesity and Vascular Research (HI-MAG), Helmholtz Zentrum Mnchen at the University of Leipzig, Leipzig, Germany, 10 CeMM Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria

doi:10.21037/jtd-21-1018 30

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