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Pro-inflammatory cytokines, activation of glial cells, and high levels of reactive oxygen species production (ROS) are known to increase nerve damage and trigger pain signaling pathways [5]

GLP-1s may influence the pathways that promote cancer not only by controlling weight and blood sugar but also through their anti-inflammatory effects independent of these traditional risk factors

6.6 Switching from Other GLP-1 Agonists or Antidiabetic Therapies Transitioning between therapies requires clinical oversight

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That mechanism helps regulate blood sugar but also increases the likelihood of gastrointestinal distress