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With the emergence of longer-acting and more potent GLP-1RAs such as semaglutide, one might assume that these compounds translate into increased efficacy in BAD compared with liraglutide

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Preclinical Brain-Protection Biology Has Not Translated Cleanly Animal and cell studies give GLP-1 drugs a plausible stroke-neuroprotection rationale

Further investigations are needed to comprehensively assess the functional and safety aspects of long-term Exe4 secretion