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Regarding mitochondrial dysfunction involved in AD pathophysiology, it includes disturbances in oxidative phosphorylation (OXPHOS), and impaired energy metabolism as well as excess generation of ROS, altered mitochondrial biogenesis, transport and dynamics (68)

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Glucagon-like Peptide-1 receptor Tie2 + cells are essential for the cardioprotective actions of liraglutide in mice with experimental myocardial infarction

.ijo201559 [DOI] [PMC free article] [PubMed] [Google Scholar] 8.Moore PW, Malone K, VanValkenburg D, Rando LL, Williams BC, Matejowsky HG, Ahmadzadeh S, Shekoohi S, Cornett EM, Kaye AD

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This phenomenon, referred to as the incretin effect, was subsequently attributed to gastrointestinal and endocrine factors