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Description
Amplifies beta-cell insulin secretion through pathways distinct from GLP-1R, modulates adipose tissue lipid handling, and appears to reduce the GI adverse-event burden associated with isolated GLP-1R agonism

Patients often report reduced food cravings and decreased interest in previously preferred foods, which may reflect altered reward processing

Every 10% reduction in body weight significantly decreases GERD symptoms and may even eliminate the need for acid-suppressing medication

Other classes of medicines used include the monoamine oxidase type B inhibitors (rasagiline

Improved Engagement Patients who feel respected and not judged are more likely to remain in treatment

Drucker, 2024)