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Role of NADPH oxidase 4 in lipopolysaccharide-induced proinflammatory responses by human aortic endothelial cells
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The latter participates in the main defense mechanisms of antioxidant against reactive oxygen species (ROS)
Trimethylamine N-oxide, the microbiome, and heart and kidney disease
Recently has been reviewed that cholesterol-mediated mitochondrial glutathione depletion is accompanied by increased Amyloid--induced oxidative stress in mitochondria (Montserrat et al
