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Resistance developed in NSCLC due to the activation of AXL receptor tyrosine kinase (AXL) and the induction of EMT in response to EGFR-targeting anticancer drugs (86)

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In ATC, glutamine metabolism and glutaminolysis are also dysregulated, with cancer cells exhibiting increased expression of glutamine transporters and enzymes involved in glutaminolysis [26]

LT was responsible for funding acquisition, project administration, and reviewing and editing the manuscript

[DOI] [PubMed] [Google Scholar] 117.Wallace JC