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567 Activation of TRPV1 by 9(10)-EpOME 622 and 12,13-DiHOME 222 resulted in increased TRPV1-dependent calcitonin gene-related peptide (CGRP)-release from sensory neurons in mice

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doi: 10.3389/fphys.2022.882792

Gain-of-function mutations disrupt this equilibrium by enhancing cation leakage and facilitating the dehydration of RBCs, resulting in hereditary xerocytosis

In addition to its healing properties with heart tissue, it appears proangiogenic, works to reduce infarct volume, preserves cardiac function in preclinical models, and is antifibrotic