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The rats in the BPC-157 group were randomly subdivided and received doses of 10, 20, or 40 g/kg BPC-157 (n = 7 in each group)

Nutritional inadequacies, enzyme defects, and pathological changes to tissues can all contribute to a reduced ability of the body to accomplish the synthesis of the active forms of vitamin B12 from CN-Cbl

[DOI] [PubMed] [Google Scholar] Broxton C

Urea cycle enzymes in human liver: ontogenesis and interaction with the synthesis of pyrimidines and polyamines

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