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Furthermore, the critical nerve gap in this model is approximately 1.5 cm, significantly less than the clinical demands, which often necessitate repairs spanning approximately 4 cm in humans
With studies characterising reduced infarct size, improved cardiac function, promotion of cardiac progenitor cell migration, cardiomyocyte survival through PI3K/Akt anti-apoptotic signalling, and stimulation of new vessel formation in infarcted myocardium
Closing Note GLP-1 agonists are changing clinical practice right now
