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TRIM32 protein modulates type I interferon induction and cellular antiviral response by targeting MITA/STING protein for K63-linked ubiquitination

doi: 10.1016/j.curtheres.2010.06.002

One of the primary mechanisms through which SPRMs exert their effects is by inhibiting progesterone-induced tyrosinase activity and the subsequent melanin synthesis pathways
& Egan, T
Kiser C, Gonul CP, Olcum M, Genc S

Gastrointestinal complications are also seen, with signs of ulceration mainly affecting the stomach and less frequently the duodenum
