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Formally, TRXR can be described as an oxidoreductase that relies on NADPH and FAD to reduce TRX-S2

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Although some people swear by its health benefits, the information regarding efficacy is insufficient to back their claims

Malfunction of SLC25A39 might interfere with synaptic dysfunction or even neurodegeneration not only in flies but also in mammals, including humans

These therapeutic ambitions are further tempered by the practical difficulties of achieving cell-type-specific modulation without disrupting homeostasis or compromising delivery across the bloodbrain barrier