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Moreover, they promptly break down glucagon-like peptides (GLP-1) and gastric inhibitory peptides (GIP), which stimulate insulin release from islet beta cells, resulting in enhancements in HOMA-IR [79]

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Genetic knock out of Cygb promotes hepatic fibrosis and the development of HCC, accompanied by increased markers of oxidative stress under the administration of diethylnitrosamine [28] or a choline-deficient amino acid-defined diet [29]

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