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Slattery K, Woods E, Zaiatz-Bittencourt V, Marks S, Chew S, Conroy M, et al

Maintained results with continued use

Therefore, the clinical benefits of these ferroptosis-targeting medications in IPF remain to be thoroughly elucidated, and their pharmacological characteristics need further refinement

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Diabetologia 50, 23742383

Besides, DMF can also prevent mitochondrial and bioenergy failure and an inflammatory imbalance to ultimately improve the clinical symptoms of X-ALD mice ( The ER stress inhibitor tauroursodeoxycholic acid (TUDCA) has been confirmed to effectively normalize several UPR responsive genes and ER stress sensors aberrantly regulated in ABCD1-mice and stop the progression of axonal and locomotor impairment in ABCD1/ABCD2 double knout out mice (109)
