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In experimental models of cerebral ischaemia, DSIP administration was associated with accelerated restoration of motor coordination and reduced neuronal damage, with proposed mechanisms involving stabilisation of neuronal membrane function and protection against excitotoxicity from excessive glutamate signalling

Mitochondria autophagy is induced after hypoxic/ischemic stress in a Drp1 dependent manner: the role of inhibition of Drp1 in ischemic brain damage

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Conversely, inhibiting microglial PKM2 ameliorated cognitive impairment and brain damage in NPSLE mice [28]

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