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*Correspondence: Michel Vignes, [email protected] Roussel, [email protected] This article was submitted to Cellular and Molecular Mechanisms of Brain-aging, a section of the journal Frontiers in Aging Neuroscience Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

[DOI] [PMC free article] [PubMed] [Google Scholar] 244.Feng W.-X., Yang Y., Wen J., Liu Y.-X., Liu L., Feng C

(7) L Pickart, J Michelle Vasquez-Soltero, and A Margolina (2012) The Human Tripeptide GHK-Cu in Prevention of Oxidative Stress and Degenerative Conditions of Aging: Implications for Cognitive Health Oxidative Medicine and Cellular Longevity, 2012 (Issue 1), Page 324832

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pylori infection does not change overall microbial richness, the infection promotes microbial dysbiosis in the small and large intestines with an increase in pro-carcinogenic and mucus-degrading bacteria and a decrease of beneficial metabolite-producers

The mechanisms are complementary rather than competitiveGLP-1 reduces energy in, while MOTS-c and 5-Amino-1MQ increase energy out