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According to Bunney et al., PLC is localized in cytoplasm in self-inhibited mode, while binding of Ras stimulates it to change its conformation and to translocate to the membrane [8]

Novel treatment targets can be classified into those that increase the number of LDL receptors (LDLRs) in hepatocytes and those that interfere with the modification and composition of atherogenic lipoproteins

doi: 10.1016/j.lfs.2005.05.103 46

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"Evidence of a four-hit mechanism involving SMARCB1 and NF2 in schwannomatosis-associated schwannomas"

Deeks SG, Collier A, Lalezari J, et al