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It is of note that this reversible growth arrest phenotype correlated with the absence of ROS accumulation in response to p16

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In colon cancer (HCT 116) cells, emodin reduces VEGF secretion and VEGFR1/2 protein expression by down-regulating Acyl-CoA synthetase long chain family member 4 (ACSL4) [468], while in liver cancer, emodin inhibits tumorigenesis through concurrently suppressing the VEGFR2-AKT-ERK1/2 signaling pathway, and inhibiting the protein expression of SMAD2/4 by enhancing miR-34a level in Hep G2 cells [469]

J Cent Nerv Syst Dis 12:1179573520962230

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This paper extends prior efforts to clarify the mechanisms underlying the protective effect of dietary energy restriction on neoplastic growth, documenting changes in gene expression within tumours as a function of host diet