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Recent studies have also shown that accumulated uremic toxins can activate AhR during CKD progression, further inhibiting mitochondrial biogenesis by promoting the ubiquitination and proteasomal degradation of PGC1-, thus accelerating renal aging and fibrosis [308]
Psychosomatic Medicine, 66(2), 276282

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Psychometric characteristics of the Italian version of the revised Fibromyalgia Impact Questionnaire using classical test theory and Rasch analysis
