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If the mitochondrial transport system becomes a bottleneck, if there is not enough carnitine to shuttle all those mobilized fatty acids into the mitochondria for burning, then some of that fat may not get oxidized efficiently

doi: 10.1161/JAHA.115.002699

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These probabilities are not presented quantitatively but were used to make qualitative probabilistic clinical inferences about effects in preference to a statistical inference based on a null-hypothesis significance test [34]

Second, the lipoyl-carnitine ester enters the matrix by facilitated diffusion through carnitine-acylcarnitine translocase (CACT) located in the inner mitochondrial membrane (31)