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Recent findings regarding miRNAs-mediated regulation of IGF-1 expression open new avenues for designing and developing nucleic acid-based therapeutic agents that can finely tune IGF-1 levels and confer protective effects in cardiovascular and metabolic disease-associated conditions
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For the treatment of liver cancer, AP may suppress tumor growth of HCC through the long non-coding RNA H19-mediated Wnt/-catenin signaling regulatory axis, which indicated that the H19 was a potential therapeutic target for HCC (Pan et al., 2021)
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