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Then, elevated ROS could affect some important gene expression, such as heme oxygenase-1 (HO-1), hypoxia-inducible factor-1 (HIF-1), and NF-B, which also aggravate oxidative stress (Lavie, 2012)

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Journal McGuire FP, et al

7c,d) reveal that Ru-hydroxide significantly downregulated pro-inflammatory mediators (TNF- and IL-1), thereby revealing the inflammatory status within the ROS-associated mandible defect region