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377,378,379 IDH1 mutations in human cancers favor consuming the NADPH for 2-HG synthesis at the expense of other NADPH-requiring pathways essential for cell viability even when NADPH is limiting

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la T4 se une a la G-actina y promueve la migracin celular y la movilizacin de clulas progenitoras, reduciendo apoptosis, inflamacin y fibrosis (Goldstein et al

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