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SHANK3 and IGF1 restore synaptic deficits in neurons from 22q13 deletion syndrome patients

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Biological individuality is such that not everybody has equivalent ability to metabolize the precursor to raise glutathione

Heres the caveat: while the animal data is overwhelmingly positive, human studies are extremely limited

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continued using Bmal1 knockout mice and reported altered clock gene expression (Cry2 and Per2) along with decreased Nrd1, Dbp, Il1 , and Nox2 expression and increased Gsr and Lpl expression, indicating that Bmal1 regulates immune response and cellular metabolism in microglia (Wang et al., 2020)