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Importantly, the observed effect cannot be explained (exclusively) as a structure change but rather as a consequence of Zn(II) dissociation due to S-thioallylation
Recent studies strongly support the notion that mitochondrial dysfunction and oxidative stress are the main drivers of OA pathogenesis 37
27 There was a 20% absolute increase in patient-perceived improvement in the short term and a 30% absolute increase in the long term
Moreover, release of nitric oxide (NO), a soluble gas produced by endothelial cells, and also endothelial-dependent hyperpolarization can negatively modulate myogenic tone by acting directly on SMCs (75)
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