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By releasing pro-inflammatory cytokines and antibodies, effector B cells in ARDS can cause tissue damage

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Rather, it may arise from a progressive erosion of anti-ferroptotic defences, including glutathione depletion, impaired lipid-repair capacity, membrane PUFA remodelling, and mitochondrial energetic failure

10.1038/nature20413 429 TejkalovH.JakobL.KvasnovS.KlaschkaJ.SechovcovH.MrzekJ.et al

Collectively, these findings suggest that distinct HIF subtypes are expressed in various synovial cell types, all of which participate in the occurrence and progression of RA