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H 2 S protects against the development of hyperglycemia-induced endothelial dysfunction by attenuating the hyperglycemia-induced enhancement of ROS formation, attenuating nuclear DNA injury, reducing the activation of the nuclear enzyme poly(ADP-ribose) polymerase, and improving cellular viability (108)

Loss of cilia, increased number of goblet cells and lamina propria edema were noticeable in this group

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