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Finally, to test whether US-based outcomes reflected regional and temporal morphological changes expected with nerve injury without repair 51,52,53 , IHC outcomes of injured ipsilateral and contralateral control nerves were inspected at each time point (Figs

The initial report was of a homozygous missense mutation in exon 15, encoding the critical SH2 domain leading to aberrant folding and aggregation of the protein

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