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Jan 15 2024;24(1):179

Renal tubule CPT1A overexpression protects from kidney fibrosis by restoring mitochondrial homeostasis

In addition, energy metabolism disorders can cause mitochondrial dysfunction, leading to an increase in ROS production and subsequently damaging the mitochondrial membrane and DNA

But on the other hand, NADPH supports ROS production via NOX2 and thereby, increased PPP and NADPH contribute to triggering ROS-dependent pro-inflammatory signaling cascades such as NF-B and MAPK (Koelwyn et al., 2018

This would be tested using the posterior probability that the beta-binomial posterior distribution from this trial was greater than the beta-binomial distribution from the historical trial

On days 1, 3, 5, and 7, DF3966A was administrated 1 h after paclitaxel