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Description
Trimethylamine N-oxide in atherogenesis: impairing endothelial self-repair capacity and enhancing monocyte adhesion
10.3945/jn.110.125633 120 ThaissC
For the current standard of care, surgeons sew the electrodes onto the heart muscle during surgery
Mechanistically, its effects cluster around vascular remodeling, nitric-oxide (NO) signaling, extracellular-matrix repair, and inflammation control
The -SH groups on these residues act as strong nucleophiles, which can conjugate with a variety of molecules, including other glutathione molecules
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